PEP-1-GLRX1 protein exhibits anti-inflammatory effects by inhibiting the activation of MAPK and NF-κB pathways in Raw 264.7 cells.

PEP-1-GLRX1 protein exhibits anti-inflammatory effects by inhibiting the activation of MAPK and NF-κB pathways in Raw 264.7 cells.

Source :

• BMB reports [ 1976-670X ] ; 2020.

Descripteurs français

• KwdFr :
  • 12-Myristate-13-acétate de phorbol (pharmacologie), Animaux (MeSH), Anti-inflammatoires (pharmacologie), Cellules RAW 264.7 (MeSH), Cyclooxygenase 2 (métabolisme), Facteur de transcription NF-kappa B (métabolisme), Glutarédoxines (pharmacologie), Inflammation (métabolisme), Inhibiteurs de la cyclooxygénase 2 (pharmacologie), Lipopolysaccharides (pharmacologie), Mercaptamine (analogues et dérivés), Mitogen-Activated Protein Kinases (antagonistes et inhibiteurs), Mitogen-Activated Protein Kinases (métabolisme), Mâle (MeSH), Nitric oxide synthase type II (antagonistes et inhibiteurs), Nitric oxide synthase type II (métabolisme), Oedème (induit chimiquement), Oedème (métabolisme), Oedème (thérapie), Peptides (MeSH), Phosphorylation (MeSH), Souris (MeSH), Souris de lignée ICR (MeSH), Transduction du signal (effets des médicaments et des substances chimiques).
• MESH :
  • analogues et dérivés : Mercaptamine.
  • antagonistes et inhibiteurs : Mitogen-Activated Protein Kinases, Nitric oxide synthase type II.
  • effets des médicaments et des substances chimiques : Transduction du signal.
  • induit chimiquement : Oedème.
  • métabolisme : Cyclooxygenase 2, Facteur de transcription NF-kappa B, Inflammation, Mitogen-Activated Protein Kinases, Nitric oxide synthase type II, Oedème.
  • pharmacologie : 12-Myristate-13-acétate de phorbol, Anti-inflammatoires, Glutarédoxines, Inhibiteurs de la cyclooxygénase 2, Lipopolysaccharides.
  • thérapie : Oedème.
  • Animaux, Cellules RAW 264.7, Mâle, Peptides, Phosphorylation, Souris, Souris de lignée ICR.

English descriptors

• KwdEn :
  • Animals (MeSH), Anti-Inflammatory Agents (pharmacology), Cyclooxygenase 2 (metabolism), Cyclooxygenase 2 Inhibitors (pharmacology), Cysteamine (analogs & derivatives), Edema (chemically induced), Edema (metabolism), Edema (therapy), Glutaredoxins (pharmacology), Inflammation (metabolism), Lipopolysaccharides (pharmacology), Male (MeSH), Mice (MeSH), Mice, Inbred ICR (MeSH), Mitogen-Activated Protein Kinases (antagonists & inhibitors), Mitogen-Activated Protein Kinases (metabolism), NF-kappa B (metabolism), Nitric Oxide Synthase Type II (antagonists & inhibitors), Nitric Oxide Synthase Type II (metabolism), Peptides (MeSH), Phosphorylation (MeSH), RAW 264.7 Cells (MeSH), Signal Transduction (drug effects), Tetradecanoylphorbol Acetate (pharmacology).
• MESH :
  • chemical , analogs & derivatives : Cysteamine.
  • chemical , antagonists & inhibitors : Mitogen-Activated Protein Kinases, Nitric Oxide Synthase Type II.
  • chemical , metabolism : Cyclooxygenase 2, Mitogen-Activated Protein Kinases, NF-kappa B, Nitric Oxide Synthase Type II.
  • chemical , pharmacology : Anti-Inflammatory Agents, Cyclooxygenase 2 Inhibitors, Glutaredoxins, Lipopolysaccharides, Tetradecanoylphorbol Acetate.
  • chemically induced : Edema.
  • drug effects : Signal Transduction.
  • metabolism : Edema, Inflammation.
  • therapy : Edema.
  • Animals, Male, Mice, Mice, Inbred ICR, Peptides, Phosphorylation, RAW 264.7 Cells.

Abstract

Affiliations:

• Corée du Sud
• Région capitale de Séoul
• Séoul

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